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SA EMS ROAD EXPERIENCEBased on HPCSA CDST v1.3 • Oct 2025
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Polytrauma

Scenario Walkthrough: Rural Polytrauma (TBI + Hemorrhagic Shock)

Scenario / Description
Case Presentation: 28-year-old male, unrestrained driver in a high-speed rollover on a regional gravel road (90-minute transport time to referral center). GCS 6 (unresponsive, snoring airway, clenching jaw), compound left femur fracture with arterial spurting, rigid/distended abdomen. Vitals: BP 78/42 (MAP 54), HR 138 (thready), RR 8/min (shallow), SpO2 84%.
DIGITAL MONITOR HUD
CALIBRATED // TRANSDUCER 01
NIBPCRIT
78/42MAP 54
HR/PULSETACHY
138SINUS PPM
ETCO2TARGET
38mmHg
SpO2Crit
84%
RESP RATEhypo
8/MIN
NEURO GCScrit
6E1V3M2
PLETH / SPO2 TRACEGAIN 1.0 • DAMPENED
DRILL STAGE 03 // CLINICAL PATHWAY

CLINICAL INTERVENTIONS & ROAD WISDOM

Step-by-step procedural execution with tactical field rationale for Case 01 under dark, rainy ground-unit conditions.

Tactical field execution
ACTIVE EXSANGUINATION

STEP 1: CIRCULATION PRIORITY

CDST P. 6, 8, 9 & 28

Tactical field execution

Universal Initial Action (Pages 6 & 28) Basic Life Support Cadres (BAA vs. Registered ECA)

Both cadres immediately apply direct pressure to the compound femoral bleed, pack with hemostatic gauze, and apply a mechanical tourniquet 5–7 cm proximal until spurting halts completely. Apply pelvic sheet/binder.

If your crew is a Legacy BAA

Ceiling (Annexure A, Page 3): BAA cannot insert an IV line and cannot administer IV fluid therapy.

The Problem: External bleed controlled, but patient remains in hypovolemic shock (BP 78/42, HR 138) from occult internal abdominal bleeding. BAA cannot treat central hypovolemia.

🚨 BAA Backup Trigger: Radio dispatch immediately: "Priority 1 trauma with occult internal hemorrhage and shock. BAA crew unable to cannulate. Requesting immediate ILS/ALS intercept on R61 while we transport."

If your crew is a Registered ECA (Higher Certificate):

Capabilities (Annexure A, Page 3 & CDST Page 29): *ECA* CAN insert a peripheral IV line (>1 year old), CAN insert an adult IO needle, and CAN initiate conservative crystalloid fluid boluses (100–250 mL IV, titrated to maintain radial pulse or MAP ≥ 65 mmHg).

The Real *ECA* Ceiling (Annexure A, Page 10 & CDST Page 9): *ECA* cannot administer Tranexamic Acid (TXA) (Orange text: ^ANT^/~ECP~ only) and cannot run inotropes/Adrenaline infusions.

🚨 *ECA* Backup Trigger: Pumping liters of crystalloids causes the lethal triad (dilutional coagulopathy, acidosis, hypothermia).

Action: *ECA* establishes IV line, runs conservative 250mL bolus, and calls dispatch for ALS/~ECP~ intercept specifically for IV Tranexamic Acid (TXA) and inotropic support while en route.

DRUG PROFILE CARD[ANT / ECP Scope Only]
STRICT CEILING: ≤ 3 HOURS POST-INJURY

Tranexamic Acid (TXA)(Antifibrinolytic Agent (Cyklokapron))

Based on HPCSA CDST v1.3 • Oct 2025
Indication:Trauma Hemorrhagic Shock • Severe Torso Hemorrhage • SBP < 90 mmHg
Dose & Route:2g IV in 100mL Saline infused over 10 minutes
Read Drug Profile
DRUG PROFILE CARD[ANT / ECP Scope Only]
TITRATION TARGET: MAP ≥ 65 mmHg

Adrenaline (Epinephrine) Inotrope / Push-Dose(Sympathomimetic Catecholamine)

Based on HPCSA CDST v1.3 • Oct 2025
Indication:Refractory Hemorrhagic Shock • Neurogenic Shock • Post-RSI Hypotension
Dose & Route:Push-Dose: 5–20 mcg IV boluses (10 mcg/mL) • Infusion: 2–10 mcg/min (titrated to MAP ≥ 65 mmHg)
Read Drug Profile

Clinical Execution on Scene (CDST Pages 8, 9 & 29 | Annexure A): Intermediate & Mid-Tier Cadres (ECA & AEA)

Vascular Access: Both cadres initiate peripheral IV access (14G/16G) in limbs/hands for patients >1 year old.

*ECA* Specific Advantage: Holds scope for Adult Intraosseous (IO) insertion if peripheral access fails.

Damage Control Fluid Titration: Administer conservative, warm crystalloid boluses of 100–250 mL IV. Recheck vitals every 10 minutes, titrating strictly to maintain a palpable radial pulse (target SBP ≥ 90 mmHg or MAP ≥ 65 mmHg) to prevent dislodging forming clots ("popping the clot").

🚨 THE SHARED ECA / AEA BACKUP TRIGGER:

The Problem: Despite IV/IO access and conservative fluid challenges, the patient has active internal bleeding and remains in refractory shock (SBP 78/42, HR 138).

The Clinical Ceiling: Neither the *ECA* nor the *AEA* can administer Tranexamic Acid (TXA) or Inotropic / Adrenaline Infusions (both marked as ^ Orange text ^ on Pages 9 & 29: ^ANT^/~ECP~ scope only).

The "Why" for Students: Pumping liters of crystalloids does not carry oxygen and actively washes out clotting factors. The patient needs TXA to halt clot breakdown (hyperfibrinolysis) and inotropic vasopressors to maintain organ perfusion.

🚨🚨🚨The Radio Action: *ECA*/*AEA* initiates rapid transport immediately while radioing for an ALS/~ECP~ intercept rendezvous: "Priority 1 polytrauma with active internal hemorrhage and refractory shock. IV access established, conservative fluids running. Requesting ALS intercept for IV Tranexamic Acid (TXA) and inotropic support en route."

Advanced Life Support & Specialist Scope (ANT / ECP)

What the ~ECP~ Delivers Upon Intercept:

Antifibrinolytic Therapy: Administers Tranexamic Acid (TXA) 2g IV over 10 minutes (Orange text, Page 29) to stabilize clot structure within the critical 3-hour post-injury window.

Chemical Hemodynamic Support: Prepares push-dose Adrenaline (5–20 mcg boluses) or titrates an Adrenaline IV infusion at 2–10 mcg/min (starting at 5 mL/hr, Page 9) to maintain MAP ≥ 65 mmHg without volume-overloading the bleeding patient.

Point-of-Care Ultrasound: Performs an on-scene eFAST exam (Page 29) to directly visualize free fluid in Morison’s pouch and the pelvis, pre-alerting the receiving regional hospital to activate the surgical trauma team and uncrossmatched blood products prior to ambulance arrival.

THE OCCULT SHOCK TRAP

External extremity hemorrhage is arrested, yet vitals reflect worsening Class IV hemorrhagic shock: BP 78/42 (MAP 54) and HR 138. Do not assume stability. Concealed intra-abdominal / retroperitoneal bleeding continues unabated.

Tactical Road Wisdom
Tactical Road Wisdom

Vascular Access in Crashing Trauma

Resus Pearls

Tactical Road Wisdom

ROAD PEARL 01TIME CEILING: 90s

The Priority Check: Is the IV Line More Important Than the Occult Bleed?

Absolutely not. Junior clinicians frequently suffer from "cannulation tunnel vision." If you spend 10 minutes on scene or on the side of a road attempting multiple IV lines while your patient’s MAP is sitting at 50 mmHg from an internal bleed, you are actively killing them. Per HPCSA CDST guidelines atempt peripheral access rapidly (maximum 2–3 attempts under 90 seconds total). If unsuccessful, do not keep digging—immediately transition to an Adult Intraosseous (IO) needle (ECA: Adult IO | ECT/ANT/ECP: All ages) or prioritize rapid transit toward surgical intervention.
Rule of Engagement: Maximum 2 attempts or 90 seconds total. If unresolved, initiate Adult Proximal Tibial/Humeral IO or commence diesel resuscitation immediately.
ROAD PEARL 02Physical Adaptation

The Clinical "BP Cuff Trick" for Collapsed Veins:

Standard elastic tourniquets roll or pinch, failing to recruit collapsed peripheral veins in the profound shock states.
Execution protocol
Inflate manual aneroid cuff to 40-50mmHg (just beneath measured diastolic pressure). This induces immense venous backpressure without occluding frail arterial inflow. Caution: Deflate cuff immediately upon cannula flush to prevent tissue ischemia and secure your line properly.
ROAD PEARL 03Sensory Drill

Palpation over Visualization: Know Your Anatomical Landmarks:

Never rely solely on what your eyes can see under dim ambulance fluorescent lighting. Crashing trauma patients are cold, vasoconstricted, and pale. Train your fingertips to palpate for venous bounce, elasticity, and depth using anatomical landmarks (e.g., the cephalic vein in the deltopectoral groove/forearm, or the basilic vein on the medial aspect).
Field Tip:
With consensus. Master your tactile anatomy during routine, non-critical transfers so that when you face profound shock in the dark, your fingers know exactly where to locate the line safely without compromising adjacent arterial or nerve structures. Practice make Perfect!
Tactical field execution
airway and neuropathway

STEP2:Airway Compromise & The Crashing Brain

CDST P. 7, 10-14

Tactical field execution

Basic Life Support Cadres (BAA vs. Registered ECA)

Execution (Pages 7 & 10): Manual in-line C-spine stabilization (jaw thrust, no head-tilt), rigid Yankauer suctioning of blood/vomitus, and gentle BVM ventilation at 1 breath every 5 seconds with 15 L/min O2.

Ceiling & Scope Trap: An OPA does not protect the trachea against pulmonary aspiration of blood and stomach acid. If the patient has trismus or intact reflexes, an OPA triggers vomiting. An *ECA* can only place an SGA in cardiac arrest (Annexure A Page 1).

🚨 BLS Backup Trigger: GCS ≤ 8 patient with intact reflexes or trismus who cannot protect their own airway. Action: Maintain basic BVM ventilations, keep airway suctioned, and call dispatch immediately for an ~ECP~ rendezvous to perform Rapid Sequence Intubation (RSI).

Intermediate & Technician Cadres (AEA / ECT / ANT)

Execution (CDST Page 11): Assess for presence of gag reflex:

If gag reflex absent: *ECT*/^ANT^ can insert a Supraglottic Airway (SGA) or perform non-drug assisted endotracheal intubation, confirming with ETCO2.

The Ceiling (Page 11 Fork): If patient has trismus, clenching, or reactive airway reflexes, forcing an airway causes severe laryngospasm, hypoxia, and fatal ICP spikes. RSI is marked as ~ Purple text ~ (~ECP~ ONLY).

🚨 ILS/*ECT* Backup Trigger: Patient needs definitive airway protection but has active muscle tone or trismus. Action: Maintain gentle oxygenation via BVM, position lateral if vomiting, and request ~ECP~ intercept for pharmacological RSI.

The Trismus and Reflex Trap

Insert an OPA into an agitated, trismic brain-injured casualty with active airway reflexes precipitates massive emesis and laryngospasm. An OPA offers ZERO protection against aspiration. Consider a NPA if midface is intact or escalate immediately to definitive ALS airway.

Emergency Care Practitioner (ECP / ALS Scope)

Execution (Pages 12–15): Executes full pre-hospital Rapid Sequence Intubation (RSI):

Hemodynamic Pre-Optimization: Administers push-dose Adrenaline (5–20 mcg) to raise MAP ≥ 65 mmHg prior to induction, preventing post-induction cardiac arrest.

Neuroprotection: Administers Fentanyl (3 mcg/kg slow IV, Page 14) to blunt the sympathomimetic reflex spike in intracranial pressure caused by direct laryngoscopy.

Induction & Paralysis: Administers reduced-dose Ketamine (1 mg/kg) or Etomidate (0.2 mg/kg), followed immediately by Rocuronium (1.2 mg/kg IV, Page 13).

Intubation & Verification: Passes cuffed ETT smoothly using a gum elastic bougie; confirms placement immediately with continuous quantitative waveform ETCO2 (35–45 mmHg).

DRUG PROFILE CARD[ECP Only (RSI)] • [ANT/ECP (Analgesia)]
POST-INDUCTION MONITORING: CONTINUOUS ETCO2

Ketamine Hydrochloride(Dissociative Anesthetic (Ketalar))

Based on HPCSA CDST v1.3 • Oct 2025
Indication:RSI Induction in Hemodynamically Unstable Trauma • Severe Neurotrauma
Dose & Route:RSI Induction: 1.0 – 2.0 mg/kg IV (Reduce to 0.5–1.0 mg/kg in shock) • Maintenance: 0.25–0.5 mg/kg/hr IV
Read Drug Profile
Tactical field execution

Advanced & Specialist Cadres (ANT / ECP Scope)

CDST P. 6, 8, 9 & 28

Tactical field execution

Emergency Care Practitioner (ECP / ALS Scope)

Execution (CDST Page 18): Connect patient to transport mechanical ventilator on lung-protective settings:

Tidal Volume: 6–8 mL/kg Ideal Body Weight (IBW).

Ventilator Rate: 12–15 breaths per minute.

PEEP: 5 cmH2O | Target Saturation: 94–98%.

Target ETCO2: Strictly maintained between 35–45 mmHg (4.6–6.0 kPa).

Post-Intubation Maintenance (Page 16 & 19): In hemodynamically unstable trauma, run continuous Ketamine (0.25–0.5 mg/kg/hr) + Midazolam (0.05 mg/kg/hr) IV infusion.

ROAD PEARL 04Sensory Drill

The "Two-Handed" Jaw Thrust for Bumpy Roads

In a shaking, dark ambulance box driving over rural gravel roads, a single-handed jaw thrust fails within 180 seconds due to severe muscle fatigue.
Tactical Adaptation:
Place your elbows firmly onto the stretcher mattress on either side of the patient's head. Place your thumbs on the patient's zygomatic arches (cheekbones) and hook your index and middle fingers beneath the angles of the mandible. Pull the jaw upward and forward dynamically. This anchors your upper body to the bed, counteracts ambulance inertia, and shifts the physical workload from your fingers to your larger forearm muscles—ensuring a patent airway for the entire transport leg.
ROAD PEARL 05Physical Adaptation

Defeating Trismus without Inducing "The Vomit Volcano"

When a brain-injured patient clenches their teeth, it is an involuntary primitive reflex driven by severe intracranial irritation. Forcing a rigid plastic Guedel OPA or metal laryngoscope blade triggers an immediate masseter spasm, laryngospasm, and an explosive vagal vomiting reflex.
Field Workaround:
Leave the jaw completely alone. Measure and lubricate a flexible Nasopharyngeal Airway (NPA). Gently pass it along the floor of the nasal cavity—only if there are no signs of mid-face trauma or basilar skull fractures (e.g., raccoon eyes, battle's sign). If the face is smashed, rely entirely on a perfect two-person BVM seal with aggressive suctioning at the lip-line until an ECP intercepts to chemically paralyze the jaw muscles.
ROAD PEARL 06Cognitive Drill

The "Finger Tapping" Cadence to Stop Hyperventilation

When a student or junior medic sees a crashing GCS 6 trauma patient with a thready pulse, their own adrenaline spikes. This causes them to unconsciously squeeze the BVM too rapidly and too deeply (the "Death Squeeze"), blowing ETCO2 into the 20s and starving the brain of oxygen.
The Cognitive Override:
To maintain a strict physiological target of 12–15 breaths per minute (1 breath every 4 to 5 seconds), use a rhythmic tactical pacing script. When you squeeze the bag, use only three fingers—never your whole hand—to deliver a small, controlled tidal volume (6–8 mL/kg). Squeeze smoothly over 1 second, then tap your index finger on the patient's collarbone while counting aloud: "Release... two... three... four... Squeeze." This mental metronome breaks the adrenaline panic loop and protects the cerebral penumbra.

WHY BAGGING TOO FAST KILLS

35 - 45 mmHg Continuous Waveform
<35 mmHg(HYPERVENTILATION)
35-45(HOMEOSTASIS)
>45 mmHg(HYPOVENTILATION)
ETC02 > 45 MMHG (HYPOVENTILATION):
Acidosis drives severe cerebral vasodilation, provoking acute intracranial pressure (ICP) surges and irreversible brainstem herniation.
ETC02 < 35 MMHG (HYPERVENTILATION):
Excess ventilation precipitates profound cerebral vasoconstriction, choking cerebral blood flow (CBF) by up to 50% and triggering extensive secondary ischemic infarction.
STRICT TARGET:35 - 45 mmHg Continuous Waveform
🧠 Clinical Insight:

Why Hyperventilation Kills the Injured Brain? 1. The Chemistry (The Trigger): Hyperventilation blows off carbon dioxide (CO2) through the lungs faster than the body produces it. As blood levels of CO2 drop (ETCO2 < 35 mmHg), the blood and cerebrospinal fluid (CSF) become alkalotic (respiratory alkalosis—a rapid rise in pH). 2. The Vascular Response (The Reaction): Cerebral blood vessels are chemically sensitive to CO2 and pH levels: High CO2 (Acidosis): Dilates cerebral vessels, increasing intracranial blood volume and spiking intracranial pressure (ICP). Low CO2 (Alkalosis): Triggers immediate, aggressive cerebral vasoconstriction—the brain’s arterial blood vessels clamp shut. 3. The Clinical Consequence (The Ischemic Trap): When cerebral arterioles constrict, Cerebral Blood Flow (CBF) plummets by 30% to 50%. In an already injured, contused, or swollen brain (TBI), this sudden drop in perfusion starves vulnerable brain tissue (the penumbra) of vital oxygen and glucose. You induce severe secondary cerebral ischemia and irreversible brain tissue death, even while your pulse oximeter shows 100% SpO2.

💡 The Golden Rule for the Road: CO2 dictates vessel caliber:

• Bagging too fast → Low ETCO2 (< 35 mmHg) → Vessels clamp shut → Brain starves of oxygen (Ischemia / Infarction). Bagging too slow → High ETCO2 (> 45 mmHg) → Vessels dilate wide → Intracranial pressure spikes (Brainstem Herniation) The Target: Maintain quantitative ETCO2 strictly between 35 and 45 mmHg at all times!

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SA EMS Field Protocols

Disclaimer: This guide is for peer discussion and educational scenario simulation only. It does not replace individual clinical judgment, statutory protocols, or local clinical governance. All images featured in this document are AI-generated conceptual illustrations and do not depict real persons, patients, or actual scenes. Not an official publication of the HPCSA or UCT.

Author: Jacoline (Jax) Geldenhuys, BEMCHPCSA Registered Emergency Care Practitioner (ECP) since 2018(Registration and credentials verified on the official HPCSA public portal)© 2026 Paramedic Cases: SA EMS Field Protocols. All rights reserved.

STATUTORY REGULATORY CONTEXT & DISCLAIMER:
This digital clinical companion is an independent educational and decision-support reference. All clinical algorithms reflect the HPCSA PBEC Clinical Decision Support Tools (CDST Version 1.3, October 2025), read in direct conjunction with the HPCSA List of Capabilities and Medications (Annexure A, April 2022) and the parent Clinical Practice Guidelines (2018).

Clinical Accountability: Per HPCSA Ethical Guidelines (Booklet 20), digital and algorithmic tools serve strictly as an adjunct to professional judgment. This platform does not provide automated diagnostic prescription. Registered practitioners are legally bound to verify all medication dosages, contraindications, and procedural scopes against their registered HPCSA scope of practice and local provincial clinical governance frameworks.